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To address this, we developed a chimeric GLP-1A that targets transporter-mediated endocytosis to enhance cellular permeability to GLP-1A by utilizing the transporters available in the intestine, particularly the apical sodium-dependent bile acid transporter (ASBT)

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HUVEC cells were fixed with 4% paraformaldehyde, permeabilized with ice-cold methanol and blocked with MinuteBlock Blocking Buffer (Affinibody, China)

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Trostchansky, A., Bonilla, L., Gonzalez-Perilli, L

Additionally, neural repression of the KCC2 could affect synaptic maturation, compromising the function of inhibitory cortical neurons (Yeo et al., 2013)